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Monoclonal Gammopathies and the Kidney
. Ed(S): Touchard, Guy (Chu La Miletrie, Poitiers, France); Aucouturier, Pierre (Hopital Tenon, Paris, France); Hermine, Olivier (Hopital Necker, Par
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Description for Monoclonal Gammopathies and the Kidney
Hardback. Divided into four parts, the first part of this book includes contributions from leading investigators in B-cell proliferations, immunoglobulin synthesis, secretion and structure, light chain uptake and metabolism in the kidney. The second and third parts focus on the tubule and the glomerulus. Editor(s): Touchard, Guy (CHU La Miletrie, Poitiers, France); Aucouturier, Pierre (Hopital Tenon, Paris, France); Hermine, Olivier (Hopital Necker, Paris, France); Ronco, Pierre (Hopital Tenon, Paris, France). Num Pages: 300 pages, biography. BIC Classification: MJR. Category: (P) Professional & Vocational; (UP) Postgraduate, Research & Scholarly; (UU) Undergraduate. Dimension: 235 x 155 x 23. Weight in Grams: 701.
Table 1 Comparison of MIDD and immunoglobulinic amyloidosis MIDD Amyloidosis Presentation Multivisceral or predominant involvement of an organ (kidney) Underlying disease Monoclonal immunoproliferative disorders, mostly myeloma. Monoclonal bone marrow plasma cell populations without overt malignancy. Possibility of 'non-secretory' forms. Deposits by EM (Granular) non-organized Fibrillar (13 pleated sheets) Spectrum LCDD : L chains, most K, either AL-amyloidosis : fragments (?)* of or apparently enlarged, normal-sized normal-sized or apparently enlarged or short. Frequent glycosylation L chains most A. Frequent and polymeration. glycosylation.Abnormal susceptibility to proteolysis. LHCDD : associated H chains Cases with associated short H chains (short by biosynthesis by biosynthesis ... Read more
Table 1 Comparison of MIDD and immunoglobulinic amyloidosis MIDD Amyloidosis Presentation Multivisceral or predominant involvement of an organ (kidney) Underlying disease Monoclonal immunoproliferative disorders, mostly myeloma. Monoclonal bone marrow plasma cell populations without overt malignancy. Possibility of 'non-secretory' forms. Deposits by EM (Granular) non-organized Fibrillar (13 pleated sheets) Spectrum LCDD : L chains, most K, either AL-amyloidosis : fragments (?)* of or apparently enlarged, normal-sized normal-sized or apparently enlarged or short. Frequent glycosylation L chains most A. Frequent and polymeration. glycosylation.Abnormal susceptibility to proteolysis. LHCDD : associated H chains Cases with associated short H chains (short by biosynthesis by biosynthesis ... Read more
Product Details
Format
Hardback
Publication date
2003
Publisher
Kluwer Academic Publishers United States
Number of pages
300
Condition
New
Number of Pages
300
Place of Publication
New York, NY, United States
ISBN
9781402010750
SKU
V9781402010750
Shipping Time
Usually ships in 15 to 20 working days
Ref
99-15
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